Human male and female sex act. Primary And Secondary Sex Organs.



Human male and female sex act

Human male and female sex act

Sexual behavior in males and females has three components: In most species, either the male or the female has evolved elaborate displays to attract the opposite sex. Without motivation, the desire to seek out sex is low. The copulatory act itself is the process of DNA exchange.

All of these aspects of sexual behavior are under hormonal control. Males A classic study by Berthold showed, in , that a hormone secreted from the testes was responsible for comb growth in roosters. The isolation of a testicular hormone, named testosterone, in opened the door for studies on the hormonal basis for reproductive behavior in males.

Once testosterone was synthesized, it was used to study male sexual behavior. Studies in the late s and early s were instrumental in showing that testosterone restores male sexual behavior. Another study, by Beach and Holtz-Tucker in ,2 demonstrated that castration followed by exogenous testosterone treatment maintained sexual behavior. These studies confirmed a critical role of testosterone in mediating male sexual behavior, and illustrated the point that the dose of testosterone needed to maintain existing sexual behavior was considerably less than the dose needed to restore it following castration.

The pattern of hormone secretion in males is tonic Fig. Males are sexually receptive as long as testosterone levels are high. Castration results in a gradual decline in sexual behavior that, in rats, takes approximately 10—14 days. Restoration of male sexual behavior by testosterone is also gradual and takes about 10—14 days.

The reason for this lengthy time for hormone action is not well understood, especially since testosterone levels fall to undetectable levels within 24 hours of castration. The more sexual experience the animal has, whether rodent or human, the longer sexual behavior is exhibited in the absence of testosterone. In animals that are seasonal breeders, testosterone levels, while basically tonic in nature, rise and fall with the seasons.

Sexual interest rises and falls in concert with hormone levels. The importance of testosterone as the male sex hormone was challenged in the s and s by studies showing that estradiol can also increase male sexual behavior in castrated males.

These earlier studies showed that DHT was minimally effective in restoring male sexual behavior. However, estradiol restored some aspects of male sexual behavior. The potential importance of estrogen was bolstered by studies showing that administration of estrogen receptor blockers inhibited male sexual behaviors. Finally, the aromatase inhibitor ATD 1,4,6-androstatriene-3,dione reportedly prevented restoration of sexual behavior in males.

The results of these experiments pointed to an important role for estrogen in the neuromodulation of sexual behavior in males. It was thought that testosterone was a prohormone and that estradiol was the active agent in stimulating male sexual behavior.

Most of the earlier studies employed high doses of estradiol. Later studies reported that silastic capsules filled with estradiol, which provided physiological levels of hormone, were ineffective in restoring male sexual behavior. The early studies using ATD were brought into question by the finding that ATD is also an effective androgen receptor blocker.

With the development of specific aromatase inhibitors that do not bind to androgen receptors, it was possible to re-examine this issue. Using the specific aromatase inhibitor fadrozole, it was found that blocking the formation of estrogen does indeed prevent the restoration of male sexual behavior by testosterone.

Females There are four epochs of time in the lives of mammalian females during which fluctuations in ovarian hormones are behaviorally important: For inducing courtship and mating behaviors in laboratory mice and rats, the optimal pattern and timing of ovarian hormone administration mimics the natural estrous cycle.

It is the cycle itself that distinguishes female-type neuroendocrinology from male-type Fig. These cycling hormones are absolutely essential for female laboratory animals to perform female-typical sexual behaviors.

A period of more-or-less constant exposure to an estrogen such as estradiol, preferably 48 hours or longer, is followed by an injection of progesterone, followed 2—5 hours later by behavioral tests.

Progesterone used this way amplifies the positive effect of estrogens on female reproductive behavior. We note that, for some social and sexual behaviors, metabolites of progesterone also play a role.

In particular, metabolites of progesterone that reduce anxiety make it easier for female laboratory animals to initiate courtship responses. Thus, the data for female sexual behaviors illustrate three principles of hormone action on the brain:

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Female Reproductive System



Human male and female sex act

Sexual behavior in males and females has three components: In most species, either the male or the female has evolved elaborate displays to attract the opposite sex. Without motivation, the desire to seek out sex is low. The copulatory act itself is the process of DNA exchange. All of these aspects of sexual behavior are under hormonal control. Males A classic study by Berthold showed, in , that a hormone secreted from the testes was responsible for comb growth in roosters.

The isolation of a testicular hormone, named testosterone, in opened the door for studies on the hormonal basis for reproductive behavior in males. Once testosterone was synthesized, it was used to study male sexual behavior. Studies in the late s and early s were instrumental in showing that testosterone restores male sexual behavior.

Another study, by Beach and Holtz-Tucker in ,2 demonstrated that castration followed by exogenous testosterone treatment maintained sexual behavior. These studies confirmed a critical role of testosterone in mediating male sexual behavior, and illustrated the point that the dose of testosterone needed to maintain existing sexual behavior was considerably less than the dose needed to restore it following castration.

The pattern of hormone secretion in males is tonic Fig. Males are sexually receptive as long as testosterone levels are high.

Castration results in a gradual decline in sexual behavior that, in rats, takes approximately 10—14 days. Restoration of male sexual behavior by testosterone is also gradual and takes about 10—14 days. The reason for this lengthy time for hormone action is not well understood, especially since testosterone levels fall to undetectable levels within 24 hours of castration.

The more sexual experience the animal has, whether rodent or human, the longer sexual behavior is exhibited in the absence of testosterone. In animals that are seasonal breeders, testosterone levels, while basically tonic in nature, rise and fall with the seasons.

Sexual interest rises and falls in concert with hormone levels. The importance of testosterone as the male sex hormone was challenged in the s and s by studies showing that estradiol can also increase male sexual behavior in castrated males.

These earlier studies showed that DHT was minimally effective in restoring male sexual behavior. However, estradiol restored some aspects of male sexual behavior. The potential importance of estrogen was bolstered by studies showing that administration of estrogen receptor blockers inhibited male sexual behaviors. Finally, the aromatase inhibitor ATD 1,4,6-androstatriene-3,dione reportedly prevented restoration of sexual behavior in males.

The results of these experiments pointed to an important role for estrogen in the neuromodulation of sexual behavior in males. It was thought that testosterone was a prohormone and that estradiol was the active agent in stimulating male sexual behavior. Most of the earlier studies employed high doses of estradiol. Later studies reported that silastic capsules filled with estradiol, which provided physiological levels of hormone, were ineffective in restoring male sexual behavior.

The early studies using ATD were brought into question by the finding that ATD is also an effective androgen receptor blocker. With the development of specific aromatase inhibitors that do not bind to androgen receptors, it was possible to re-examine this issue. Using the specific aromatase inhibitor fadrozole, it was found that blocking the formation of estrogen does indeed prevent the restoration of male sexual behavior by testosterone.

Females There are four epochs of time in the lives of mammalian females during which fluctuations in ovarian hormones are behaviorally important: For inducing courtship and mating behaviors in laboratory mice and rats, the optimal pattern and timing of ovarian hormone administration mimics the natural estrous cycle.

It is the cycle itself that distinguishes female-type neuroendocrinology from male-type Fig. These cycling hormones are absolutely essential for female laboratory animals to perform female-typical sexual behaviors. A period of more-or-less constant exposure to an estrogen such as estradiol, preferably 48 hours or longer, is followed by an injection of progesterone, followed 2—5 hours later by behavioral tests. Progesterone used this way amplifies the positive effect of estrogens on female reproductive behavior.

We note that, for some social and sexual behaviors, metabolites of progesterone also play a role. In particular, metabolites of progesterone that reduce anxiety make it easier for female laboratory animals to initiate courtship responses. Thus, the data for female sexual behaviors illustrate three principles of hormone action on the brain:

Human male and female sex act

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5 Comments

  1. Flattened anterior to posterior, the endocervical canal measures seven to eight mm at its widest in reproductive-aged women. This behavior is characterized by a fixation on sexual intercourse and disinhibition.

  2. Because the urethra is so close to the anus, women should always wipe themselves from front to back to avoid infecting the vagina and urethra with bacteria. Labia majora Outer skin folds that surround the entrance to the vagina. If testosterone levels are low, hormone replacement therapy may help relieve symptoms, such as the loss of interest in sex, depression, and fatigue.

  3. The term "vagina" is often improperly used as a generic term to refer to the vulva or female genitals, even though - strictly speaking - the vagina is a specific internal structure and the vulva is the exterior genitalia only. Factors such as biological and psychological factors, diseases, mental conditions, boredom with the relationship, and widowhood have been found to contribute with the common decrease in sexual interest and activity in old age.

  4. The belts were worn by women to protect their chastity, which included preventing masturbation or sexual access by unauthorized males.

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